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The existence of NTG unequivocally underscores the necessity to investigate pressure-independent mechanisms, with compelling evidence now positioning ferroptosis as a pivotal driver of RGC loss in glaucoma [1]

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Using the LPS-induced inflammatory PE rat model, we demonstrated that oral pre-treatment with NR prevents the development of PE-like clinical features, evidenced by lowered maternal blood pressure, decreased placental inflammation, increased placental and fetal weights, and increased fetal survival