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Senescent cell SASP (senescence-associated secretory phenotype) suppression is an emerging GHK-Cu research area: GHK-Cu reduces SA-β-galactosidase activity (senescence marker), p16/p21 expression, and SASP cytokine secretion (IL-6, IL-8, GDF15) in hydrogen peroxide-induced premature senescence models — mechanistically through NF-κB suppression and p53 pathway modulation
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