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A key advantage of this model is the ability to study the function of innate immune biology effects on the pathophysiology of demyelination/remyelination in the CNS without confounding peripheral infiltrates such as T-cells (Praet et al., 2014, Hillis et al., 2016)

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This supposition is based on our recent demonstration that, at least in rat hippocampal neurons, c-Met is premultimerized at postsynaptic densities as dimers, tetramers, hexamers, and octomers in the absence of HGF (Kawas et al., 2013)