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Compared with control cells, the mRNAs of ACC (acetyl-CoA carboxylase), CHOP (CCAAT/enhancer-binding protein homologous protein) and caspase -3 significantly increased and PPAR -γ (peroxisome proliferator activated receptor γ) mRNAs significantly decreased in the alcohol-induced model groups of human normal liver cell line LO2 (Figure 7A), hepatocellular carcinoma cell line HepG2 (Figure 7B) and model animal (Figure 7C)

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GLP-1 RAs inhibit the TLR4/MYD88/NF-κB signaling pathway in renal tubular cells, suppressing NLRP3 inflammasome activation and subsequent IL-1β/IL-18 release, which are key drivers of interstitial inflammation (102)