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A likely mechanism for this effect is the ability of fructose to reduce nitric oxide (NO) availability due to uncoupling of endothelia nitric oxide synthase (eNOS), as NO donors mitigated the fructose-induced ICAM-1 expression [43–45]
Immunité
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Efruxifermin combined with a GLP-1 receptor agonist reduces liver fat in NASH
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